Obesity and Cancer: Mechanisms, Epidemiological Evidence, and Potential Risk Reduction.

  • Reading time:2 mins read

Journal: Journal of obesity & metabolic syndrome

This narrative review synthesizes recent meta-analyses and key cohort data linking excess adiposity to cancer incidence, focusing on body mass index and waist circumference.

Key points:

  • Across the 13 malignancies recognized as obesity-related, higher adiposity is consistently associated with increased cancer risk. Effect sizes vary by:
    • Histologic subtype (e.g., stronger for esophageal adenocarcinoma than squamous cell carcinoma)
    • Anatomic subsite (e.g., gastric cardia vs non-cardia)
    • Sex and menopausal status
    • Adiposity metric, with central adiposity (waist circumference) often conferring additional risk beyond BMI.
  • Evidence is heterogeneous or less conclusive for certain cancers:
    • Mixed or less consistent data for pancreas and thyroid.
    • Emerging but not firmly established associations for oral cavity, melanoma, bladder, non-Hodgkin lymphoma, and leukemia.
  • Prevention and weight loss:
    • Observational studies and selected interventional data suggest that intentional weight loss—via lifestyle change, pharmacotherapy (including GLP-1 receptor agonists), or bariatric surgery—may reduce overall or site‑specific cancer incidence.
    • Effect estimates are variable, and residual confounding and causality remain concerns.
  • Overall conclusion:
    • Convergent epidemiologic data and underlying biologic plausibility support obesity as a modifiable risk factor for multiple cancers.
    • The authors call for standardized adiposity measures, consistent stratification, better confounder control, and adequately powered long-term randomized or quasi-experimental studies to refine effect estimates and enable more precise cancer prevention strategies in people with obesity.

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